Why is that?
They discover these rodents stored calories in fat tissues as opposed to the other tissues that burn the calories and use them as energy. This causes the glucose to produce even more fat, which is unusual. Normally, a special membrane works as a door to keep the glucose from spreading to other cells, but the mutation keeps the “flood gates opened.”
"We found that mice can become obese without eating more, and that there is an underlying cellular mechanism to explain that weight gain," Bennett said. "This gene could enable us to identify at-risk individuals who should watch what kind of calories they eat and exercise more in order to keep their body weight under control."
For future studies, researchers hope to identify humans with the gene to determine how it could affect other variants of health.
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